Pseudoexon activation in disease by non-splice site deep intronic sequence variation — wild type pseudoexons constitute high-risk sites in the human genome

Ulrika S.S. Petersen, Thomas K. Doktor, Brage S. Andresen*

*Corresponding author for this work

Research output: Contribution to journalJournal articleResearchpeer-review

Fingerprint

Dive into the research topics of 'Pseudoexon activation in disease by non-splice site deep intronic sequence variation — wild type pseudoexons constitute high-risk sites in the human genome'. Together they form a unique fingerprint.

Medicine & Life Sciences