Sodium retention by uPA-plasmin-ENaC in nephrotic syndrome-Authors reply

Gitte R Hinrichs, Kathrin Weyer, Ulla G Friis, Per Svenningsen, Ida Katrine Lund, Rikke Nielsen, Géraldine Mollet, Corinne Antignac, Claus Bistrup, Boye L Jensen, Henrik Birn

Publikation: Bidrag til tidsskriftKommentar/debatForskningpeer review

Abstrakt

There is a great interest in exploring the mechanisms of oedema in nephrotic syndrome (NS) as evidenced by 2 parallel studies published in Acta Physiologica (1, 2). This has generated an editorial comment (3) which we respond to by this rebuttal. The interest in the nephrotic edema reflects not only the significant negative impact this has on the quality of life, but also the difficulties associated with identifying optimal treatment due to the lack of pathophysiological insight.

OriginalsprogEngelsk
Artikelnummere13432
TidsskriftActa Physiologica (Print)
Vol/bind228
Udgave nummer4
ISSN1748-1708
DOI
StatusUdgivet - apr. 2020

Bibliografisk note

© 2019 Scandinavian Physiological Society. Published by John Wiley & Sons Ltd.

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