Transient receptor potential (TRP) channel-induced cation influx activates human monocytes, which play an important role in the pathogenesis of atherosclerosis. In the present study, we investigated the effects of high glucose-induced oxidative stress on TRP channel expression in human monocytes.
Wuensch, T., Thilo, F., Krueger, K., Scholze, A., Ristow, M., & Tepel, M. (2010). High glucose-induced oxidative stress increases transient receptor potential channel expression in human monocytes. Diabetes, 59(4), 844-9. https://doi.org/10.2337/db09-1100